[308] Shi Y., Pulliam D.A., Liu Y., Hamilton R.T., Jernigan A.L., Bhattacharya A., Sloane L.B., Qi W., Chaudhuri A., Buffenstein R., Ungvari Z., Austad S.N., Van Remmen H. (2013) Reduced mitochondrial ROS, enhanced antioxidant defense, and distinct age-related changes in oxidative damage in muscles of long-lived
[309] Shipounova I.N., Svinareva D.A., Petrova T.V., Lyamzaev K.G., Chernyak B.V., Drize N.I., Skulachev V.P. (2010) Reactive oxygen species produced in mitochondria are involved in age-dependent changes of hematopoietic and mesenchymal progenitor cells in mice. A study with the novel mitochondria-targeted antioxidant SkQ1.
[310] Shirey K.A., Lai W., Scott A.J., Lipsky M., Mistry P., Pletneva L.M., Karp C.L., McAlees J., Gioannini T.L., Weiss J., Chen W.H., Ernst R.K., Rossignol D.P., Gusovsky F., Blanco J.C., Vogel S.N. (2013) The TLR4 antagonist Eritoran protects mice from lethal influenza infection.
[311] Shringarpure R., Davies K.J. (2002) Protein turnover by the proteasome in aging and disease.
[312] Silachev D.N., Isaev N.K., Pevzner I.B., Zorova L.D., Stelmashook E.V., Novikova S.V., Plotnikov E.Y., Skulachev V.P., Zorov D.B. (2012) The mitochondria-targeted antioxidants and remote kidney preconditioning ameliorate brain damage through kidney-to-brain cross-talk.
[313] Sitte N., Merker K., Von Zglinicki T., Grune T., Davies K.J. (2000) Protein oxidation and degradation during cellular senescence of human BJ fibroblasts: part I-effects of proliferative senescence.
[314] Sivan S.S., Van El B., Merkher Y., Schmelzer C.E., Zuurmond A.M., Heinz A., Wachtel E., Varga P.P., Lazary A., Brayda-Bruno M., Maroudas A. (2012) Longevity of elastin in human intervertebral disc as probed by the racemization of aspartic acid.
[315] Skulachev M.V., Antonenko Y.N., Anisimov V.N., Chernyak B.V., Cherepanov D.A., Chistyakov V.A., Egorov M.V., Kolosova N.G., Korshunova G.A., Lyamzaev K.G., Plotnikov E.Y., Roginsky V.A., Savchenko A.Y., Severina I.I., Severin F.F., Shkurat T.P., Tashlitsky V.N., Shidlovsky K.M., Vyssokikh M.Y., Zamyatnin A.A., Zorov D.B., Skulachev V.P. (2011) Mitochondrial-targeted plastoquinone derivatives. Effect on senescence and acute age-related pathologies.
[316] Skulachev V.P. (1988) Membrane bioenergetics, Springer-Verlag. Berlin; New York.
[317] Skulachev V.P. (1991) Fatty acid circuit as a physiological mechanism of uncoupling of oxidative phosphorylation.
[318] Skulachev V.P. (1996) Role of uncoupled and non-coupled oxidations in maintenance of safely low levels of oxygen and its one-electron reductants.
[319] Skulachev V.P. (1996) Why are mitochondria involved in apoptosis? Permeability transition pores and apoptosis as selective mechanisms to eliminate superoxide-producing mitochondria and cell.
[320] Skulachev V.P. (1998) Uncoupling: new approaches to an old problem of bioenergetics.
[321] Skulachev V.P. (2002) Programmed death in yeast as adaptation?
[322] Skulachev V.P. (2002) Programmed death phenomena: from organelle to organism.
[323] Skulachev V.P. (2003) Aging and the programmed death phenomena. Topics Curr Genet, Model Systems in Aging (Nystrom, T. and Osiewacz, H.D.), 192–237. Springer-Verlag, Berlin Heidelberg.
[324] Skulachev V.P. (2011) Aging as a particular case of phenoptosis, the programmed death of an organism (A response to Kirkwood and Melov "On the programmed/non-programmed nature of ageing within the life history").
[325] Skulachev V.P. (2011) SkQ1 treatment and food restriction — two ways to retard an aging program of organisms.
[326] Skulachev V.P. (2012) Mitochondria-targeted antioxidants as promising drugs for treatment of age-related brain diseases.
[327] Skulachev V.P. (2013) Cationic antioxidants as a powerful tool against mitochondrial oxidative stress.
